Please use this identifier to cite or link to this item: https://hdl.handle.net/11499/37448
Title: Is the Reason of Increased D-Dimer Levels in COVID-19 Because of ACE-2-Induced Apoptosis in Endothelium?
Authors: Güler, Nil
Siddiqui, F.
Fareed, J.
Keywords: angiotensin converting enzyme 2
antithrombin
D dimer
fibrin degradation product
angiotensin II
caspase inhibitor
dipeptidyl carboxypeptidase
fibrin fragment D
virus receptor
adult respiratory distress syndrome
apoptosis
Avian infectious bronchitis virus
blood clotting disorder
Coronavirinae
coronavirus disease 2019
disease severity
endothelium
human
intensive care unit
Letter
microenvironment
pathogenesis
priority journal
Swine acute diarrhea syndrome coronavirus
thrombin time
virus pneumonia
Zika virus
Alphacoronavirus
animal
Betacoronavirus
biological model
blood
Chikungunya virus
complication
Coronavirus infection
drug effect
enzymology
host pathogen interaction
metabolism
Orthomyxoviridae
pandemic
pathogenicity
pathology
physiology
pig
thrombophilia
vascular endothelium
virology
virus replication
Angiotensin II
Animals
Apoptosis
Caspase Inhibitors
Coronavirus Infections
Endothelium, Vascular
Fibrin Fibrinogen Degradation Products
Host-Pathogen Interactions
Humans
Models, Cardiovascular
Pandemics
Peptidyl-Dipeptidase A
Pneumonia, Viral
Receptors, Virus
Swine
Thrombophilia
Virus Replication
Zika Virus
Publisher: SAGE Publications Inc.
Abstract: Abstract Not Available
URI: https://hdl.handle.net/11499/37448
https://doi.org/10.1177/1076029620935526
ISSN: 1076-0296
Appears in Collections:PubMed İndeksli Yayınlar Koleksiyonu / PubMed Indexed Publications Collection
Scopus İndeksli Yayınlar Koleksiyonu / Scopus Indexed Publications Collection
Tıp Fakültesi Koleksiyonu
WoS İndeksli Yayınlar Koleksiyonu / WoS Indexed Publications Collection

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