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https://hdl.handle.net/11499/37448| Title: | Is the Reason of Increased D-Dimer Levels in COVID-19 Because of ACE-2-Induced Apoptosis in Endothelium? | Authors: | Güler, Nil Siddiqui, F. Fareed, J. |
Keywords: | angiotensin converting enzyme 2 antithrombin D dimer fibrin degradation product angiotensin II caspase inhibitor dipeptidyl carboxypeptidase fibrin fragment D virus receptor adult respiratory distress syndrome apoptosis Avian infectious bronchitis virus blood clotting disorder Coronavirinae coronavirus disease 2019 disease severity endothelium human intensive care unit Letter microenvironment pathogenesis priority journal Swine acute diarrhea syndrome coronavirus thrombin time virus pneumonia Zika virus Alphacoronavirus animal Betacoronavirus biological model blood Chikungunya virus complication Coronavirus infection drug effect enzymology host pathogen interaction metabolism Orthomyxoviridae pandemic pathogenicity pathology physiology pig thrombophilia vascular endothelium virology virus replication Angiotensin II Animals Apoptosis Caspase Inhibitors Coronavirus Infections Endothelium, Vascular Fibrin Fibrinogen Degradation Products Host-Pathogen Interactions Humans Models, Cardiovascular Pandemics Peptidyl-Dipeptidase A Pneumonia, Viral Receptors, Virus Swine Thrombophilia Virus Replication Zika Virus |
Publisher: | SAGE Publications Inc. | Abstract: | Abstract Not Available | URI: | https://hdl.handle.net/11499/37448 https://doi.org/10.1177/1076029620935526 |
ISSN: | 1076-0296 |
| Appears in Collections: | PubMed İndeksli Yayınlar Koleksiyonu / PubMed Indexed Publications Collection Scopus İndeksli Yayınlar Koleksiyonu / Scopus Indexed Publications Collection Tıp Fakültesi Koleksiyonu WoS İndeksli Yayınlar Koleksiyonu / WoS Indexed Publications Collection |
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| File | Size | Format | |
|---|---|---|---|
| 1076029620935526.pdf | 139.14 kB | Adobe PDF | View/Open |
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